Hyper-IgE syndrome

Hidetoshi Takada, Akihiko Nomura, Toshiro Hara

Research output: Contribution to journalArticle

2 Citations (Scopus)

Abstract

Hyperimmunoglobulin-E syndrome is one of the primary immunodeficiency with the manifestations of recurrent infections especially with Staphylococcus aureus, characteristic facies, hyperextensibility of joints, multiple bone fractures, scoliosis, and delayed shedding of the primary teeth. It is a multisystem disease of autosomal dominant inheritance. Recently, a new type of hyper-IgE syndrome with autosomal recessive inheritance was identified. Although Th1/Th2 imbalance has been suspected to be a cause of this diesease, it is not clarified yet.

Original languageEnglish
Pages (from-to)361-366
Number of pages6
JournalJapanese Journal of Clinical Immunology
Volume27
Issue number6
DOIs
Publication statusPublished - Jan 1 2004

Fingerprint

Job Syndrome
Deciduous Tooth
Bone Fractures
Scoliosis
Staphylococcus aureus
Joints
Infection
Multiple Fractures

All Science Journal Classification (ASJC) codes

  • Immunology and Allergy
  • Immunology

Cite this

Takada, H., Nomura, A., & Hara, T. (2004). Hyper-IgE syndrome. Japanese Journal of Clinical Immunology, 27(6), 361-366. https://doi.org/10.2177/jsci.27.361

Hyper-IgE syndrome. / Takada, Hidetoshi; Nomura, Akihiko; Hara, Toshiro.

In: Japanese Journal of Clinical Immunology, Vol. 27, No. 6, 01.01.2004, p. 361-366.

Research output: Contribution to journalArticle

Takada, H, Nomura, A & Hara, T 2004, 'Hyper-IgE syndrome', Japanese Journal of Clinical Immunology, vol. 27, no. 6, pp. 361-366. https://doi.org/10.2177/jsci.27.361
Takada, Hidetoshi ; Nomura, Akihiko ; Hara, Toshiro. / Hyper-IgE syndrome. In: Japanese Journal of Clinical Immunology. 2004 ; Vol. 27, No. 6. pp. 361-366.
@article{0d8928e5a7214ea3b391daf74d2573db,
title = "Hyper-IgE syndrome",
abstract = "Hyperimmunoglobulin-E syndrome is one of the primary immunodeficiency with the manifestations of recurrent infections especially with Staphylococcus aureus, characteristic facies, hyperextensibility of joints, multiple bone fractures, scoliosis, and delayed shedding of the primary teeth. It is a multisystem disease of autosomal dominant inheritance. Recently, a new type of hyper-IgE syndrome with autosomal recessive inheritance was identified. Although Th1/Th2 imbalance has been suspected to be a cause of this diesease, it is not clarified yet.",
author = "Hidetoshi Takada and Akihiko Nomura and Toshiro Hara",
year = "2004",
month = "1",
day = "1",
doi = "10.2177/jsci.27.361",
language = "English",
volume = "27",
pages = "361--366",
journal = "Immunological Medicine",
issn = "0911-4300",
publisher = "Taylor and Francis Ltd.",
number = "6",

}

TY - JOUR

T1 - Hyper-IgE syndrome

AU - Takada, Hidetoshi

AU - Nomura, Akihiko

AU - Hara, Toshiro

PY - 2004/1/1

Y1 - 2004/1/1

N2 - Hyperimmunoglobulin-E syndrome is one of the primary immunodeficiency with the manifestations of recurrent infections especially with Staphylococcus aureus, characteristic facies, hyperextensibility of joints, multiple bone fractures, scoliosis, and delayed shedding of the primary teeth. It is a multisystem disease of autosomal dominant inheritance. Recently, a new type of hyper-IgE syndrome with autosomal recessive inheritance was identified. Although Th1/Th2 imbalance has been suspected to be a cause of this diesease, it is not clarified yet.

AB - Hyperimmunoglobulin-E syndrome is one of the primary immunodeficiency with the manifestations of recurrent infections especially with Staphylococcus aureus, characteristic facies, hyperextensibility of joints, multiple bone fractures, scoliosis, and delayed shedding of the primary teeth. It is a multisystem disease of autosomal dominant inheritance. Recently, a new type of hyper-IgE syndrome with autosomal recessive inheritance was identified. Although Th1/Th2 imbalance has been suspected to be a cause of this diesease, it is not clarified yet.

UR - http://www.scopus.com/inward/record.url?scp=17144393289&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=17144393289&partnerID=8YFLogxK

U2 - 10.2177/jsci.27.361

DO - 10.2177/jsci.27.361

M3 - Article

C2 - 15678888

AN - SCOPUS:17144393289

VL - 27

SP - 361

EP - 366

JO - Immunological Medicine

JF - Immunological Medicine

SN - 0911-4300

IS - 6

ER -