TY - JOUR
T1 - Intimal thickening and the distribution of vasomotor nerves in the mechanically injured dog coronary artery
AU - Taguchi, Tomoaki
AU - Ishii, Yutaka
AU - Matsubara, Fujio
AU - Tanaka, Kenzo
PY - 1986/4
Y1 - 1986/4
N2 - Intimal injury and atherosclerotic change seem to be causative factors linked to spasm of the coronary artery. Intimal thickening was produced by mechanical injury to the endothelium of the canine coronary artery and we investigated the distribution of adrenergic, cholinergic, and peptidergic nerves in the coronary arteries. Although adrenergic and cholinergic nerves were not altered in density, neuron specific enolase positive nerve fibers were increased in number in dogs killed 1 and 3 months after injury. Substance P-containing fibers were also increased at 3 months after the induced injury.
AB - Intimal injury and atherosclerotic change seem to be causative factors linked to spasm of the coronary artery. Intimal thickening was produced by mechanical injury to the endothelium of the canine coronary artery and we investigated the distribution of adrenergic, cholinergic, and peptidergic nerves in the coronary arteries. Although adrenergic and cholinergic nerves were not altered in density, neuron specific enolase positive nerve fibers were increased in number in dogs killed 1 and 3 months after injury. Substance P-containing fibers were also increased at 3 months after the induced injury.
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U2 - 10.1016/0014-4800(86)90064-X
DO - 10.1016/0014-4800(86)90064-X
M3 - Article
C2 - 2422054
AN - SCOPUS:0022476546
SN - 0014-4800
VL - 44
SP - 138
EP - 146
JO - Experimental and Molecular Pathology
JF - Experimental and Molecular Pathology
IS - 2
ER -