Neurodegeneration caused by accumulation of an oxidized base lesion, 8-oxoguanine, in nuclear and mitochondrial DNA: from animal models to human diseases.

Research output: Chapter in Book/Report/Conference proceedingChapter

Original languageEnglish
Title of host publicationThe Base Excision Repair Pathway: Molecular Mechanisms and Role in Disease Development and Therapeutic Design
PublisherWorld Scientific Publishing Co. Pte Ltd
Chapter14
Pages523-556
Number of pages34
ISBN (Print)978-981-4719-74-2
Publication statusPublished - Apr 1 2017

Cite this

Nakabeppu, Y. (2017). Neurodegeneration caused by accumulation of an oxidized base lesion, 8-oxoguanine, in nuclear and mitochondrial DNA: from animal models to human diseases. In The Base Excision Repair Pathway: Molecular Mechanisms and Role in Disease Development and Therapeutic Design (pp. 523-556). World Scientific Publishing Co. Pte Ltd.

Neurodegeneration caused by accumulation of an oxidized base lesion, 8-oxoguanine, in nuclear and mitochondrial DNA: from animal models to human diseases. / Nakabeppu, Yusaku.

The Base Excision Repair Pathway: Molecular Mechanisms and Role in Disease Development and Therapeutic Design . World Scientific Publishing Co. Pte Ltd, 2017. p. 523-556.

Research output: Chapter in Book/Report/Conference proceedingChapter

Nakabeppu, Y 2017, Neurodegeneration caused by accumulation of an oxidized base lesion, 8-oxoguanine, in nuclear and mitochondrial DNA: from animal models to human diseases. in The Base Excision Repair Pathway: Molecular Mechanisms and Role in Disease Development and Therapeutic Design . World Scientific Publishing Co. Pte Ltd, pp. 523-556.
Nakabeppu Y. Neurodegeneration caused by accumulation of an oxidized base lesion, 8-oxoguanine, in nuclear and mitochondrial DNA: from animal models to human diseases. In The Base Excision Repair Pathway: Molecular Mechanisms and Role in Disease Development and Therapeutic Design . World Scientific Publishing Co. Pte Ltd. 2017. p. 523-556
Nakabeppu, Yusaku. / Neurodegeneration caused by accumulation of an oxidized base lesion, 8-oxoguanine, in nuclear and mitochondrial DNA: from animal models to human diseases. The Base Excision Repair Pathway: Molecular Mechanisms and Role in Disease Development and Therapeutic Design . World Scientific Publishing Co. Pte Ltd, 2017. pp. 523-556
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