Abstract
In nontraumatic osteonecrosis (ON) of the femoral head, several abnormalities in the coagulation-fi brinolysis system have been reported, including increase of plasminogen activator inhibitor-1 level [ 1 ], the presence of antiphospholipid antibodies [ 2 ], familial heterozygous protein S defi ciency [ 3 ], and antithrombin III defi ciency [ 4 ]. These abnormalities are thought to induce hypercoagulability in the blood vessels.
| Original language | English |
|---|---|
| Title of host publication | Osteonecrosis |
| Publisher | Springer Berlin Heidelberg |
| Pages | 249-253 |
| Number of pages | 5 |
| ISBN (Electronic) | 9783642357671 |
| ISBN (Print) | 9783642357664 |
| DOIs | |
| Publication status | Published - Jan 1 2014 |
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All Science Journal Classification (ASJC) codes
- Medicine(all)
Cite this
The role of coagulopathy in the pathogenesis and prevention of corticosteroid-induced osteonecrosis. / Yamamoto, Takuaki; motomura, goro; Yamaguchi, Ryosuke; Iwamoto, Yukihide.
Osteonecrosis. Springer Berlin Heidelberg, 2014. p. 249-253.Research output: Chapter in Book/Report/Conference proceeding › Chapter
}
TY - CHAP
T1 - The role of coagulopathy in the pathogenesis and prevention of corticosteroid-induced osteonecrosis
AU - Yamamoto, Takuaki
AU - motomura, goro
AU - Yamaguchi, Ryosuke
AU - Iwamoto, Yukihide
PY - 2014/1/1
Y1 - 2014/1/1
N2 - In nontraumatic osteonecrosis (ON) of the femoral head, several abnormalities in the coagulation-fi brinolysis system have been reported, including increase of plasminogen activator inhibitor-1 level [ 1 ], the presence of antiphospholipid antibodies [ 2 ], familial heterozygous protein S defi ciency [ 3 ], and antithrombin III defi ciency [ 4 ]. These abnormalities are thought to induce hypercoagulability in the blood vessels.
AB - In nontraumatic osteonecrosis (ON) of the femoral head, several abnormalities in the coagulation-fi brinolysis system have been reported, including increase of plasminogen activator inhibitor-1 level [ 1 ], the presence of antiphospholipid antibodies [ 2 ], familial heterozygous protein S defi ciency [ 3 ], and antithrombin III defi ciency [ 4 ]. These abnormalities are thought to induce hypercoagulability in the blood vessels.
UR - http://www.scopus.com/inward/record.url?scp=84955744356&partnerID=8YFLogxK
UR - http://www.scopus.com/inward/citedby.url?scp=84955744356&partnerID=8YFLogxK
U2 - 10.1007/978-3-642-35767-1_33
DO - 10.1007/978-3-642-35767-1_33
M3 - Chapter
AN - SCOPUS:84955744356
SN - 9783642357664
SP - 249
EP - 253
BT - Osteonecrosis
PB - Springer Berlin Heidelberg
ER -