Itch in Atopic Dermatitis

研究成果: ジャーナルへの寄稿評論記事

12 引用 (Scopus)

抄録

Chronic itch in inflammatory skin diseases, such as atopic dermatitis, markedly diminishes the quality of life of affected individuals. Comprehensive progress has been made in understanding itch signaling and associated mediators in the skin, dorsal root ganglia, spinal cord, and central nervous system, which may amplify or suppress atopic itch. Conventional therapies for atopic dermatitis are capable of reducing atopic itch; however, most patients are not satisfied with the antipruritic capacity of conventional treatments. Exploring itch pathways and mechanisms may lead to novel therapeutic approaches for atopic itch.

元の言語英語
ページ(範囲)113-122
ページ数10
ジャーナルImmunology and Allergy Clinics of North America
37
発行部数1
DOI
出版物ステータス出版済み - 2 1 2017

Fingerprint

Atopic Dermatitis
Antipruritics
Spinal Ganglia
Skin Diseases
Spinal Cord
Therapeutics
Central Nervous System
Quality of Life
Skin

All Science Journal Classification (ASJC) codes

  • Immunology and Allergy
  • Immunology

これを引用

Itch in Atopic Dermatitis. / Kido-Nakahara, Makiko; Furue, Masutaka; Ulzii, Dugarmaa; Nakahara, Takeshi.

:: Immunology and Allergy Clinics of North America, 巻 37, 番号 1, 01.02.2017, p. 113-122.

研究成果: ジャーナルへの寄稿評論記事

@article{264b601bc3724cab961975e6318da6e0,
title = "Itch in Atopic Dermatitis",
abstract = "Chronic itch in inflammatory skin diseases, such as atopic dermatitis, markedly diminishes the quality of life of affected individuals. Comprehensive progress has been made in understanding itch signaling and associated mediators in the skin, dorsal root ganglia, spinal cord, and central nervous system, which may amplify or suppress atopic itch. Conventional therapies for atopic dermatitis are capable of reducing atopic itch; however, most patients are not satisfied with the antipruritic capacity of conventional treatments. Exploring itch pathways and mechanisms may lead to novel therapeutic approaches for atopic itch.",
author = "Makiko Kido-Nakahara and Masutaka Furue and Dugarmaa Ulzii and Takeshi Nakahara",
year = "2017",
month = "2",
day = "1",
doi = "10.1016/j.iac.2016.08.007",
language = "English",
volume = "37",
pages = "113--122",
journal = "Immunology and Allergy Clinics of North America",
issn = "0889-8561",
publisher = "W.B. Saunders Ltd",
number = "1",

}

TY - JOUR

T1 - Itch in Atopic Dermatitis

AU - Kido-Nakahara, Makiko

AU - Furue, Masutaka

AU - Ulzii, Dugarmaa

AU - Nakahara, Takeshi

PY - 2017/2/1

Y1 - 2017/2/1

N2 - Chronic itch in inflammatory skin diseases, such as atopic dermatitis, markedly diminishes the quality of life of affected individuals. Comprehensive progress has been made in understanding itch signaling and associated mediators in the skin, dorsal root ganglia, spinal cord, and central nervous system, which may amplify or suppress atopic itch. Conventional therapies for atopic dermatitis are capable of reducing atopic itch; however, most patients are not satisfied with the antipruritic capacity of conventional treatments. Exploring itch pathways and mechanisms may lead to novel therapeutic approaches for atopic itch.

AB - Chronic itch in inflammatory skin diseases, such as atopic dermatitis, markedly diminishes the quality of life of affected individuals. Comprehensive progress has been made in understanding itch signaling and associated mediators in the skin, dorsal root ganglia, spinal cord, and central nervous system, which may amplify or suppress atopic itch. Conventional therapies for atopic dermatitis are capable of reducing atopic itch; however, most patients are not satisfied with the antipruritic capacity of conventional treatments. Exploring itch pathways and mechanisms may lead to novel therapeutic approaches for atopic itch.

UR - http://www.scopus.com/inward/record.url?scp=84996844669&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=84996844669&partnerID=8YFLogxK

U2 - 10.1016/j.iac.2016.08.007

DO - 10.1016/j.iac.2016.08.007

M3 - Review article

C2 - 27886901

AN - SCOPUS:84996844669

VL - 37

SP - 113

EP - 122

JO - Immunology and Allergy Clinics of North America

JF - Immunology and Allergy Clinics of North America

SN - 0889-8561

IS - 1

ER -