Spectrum of molecular defects in juvenile myelomonocytic leukaemia includes ASXL1 mutations: Short report

Yuka Sugimoto, Hideki Muramatsu, Hideki Makishima, Courtney Prince, Anna M. Jankowska, Nao Yoshida, Yinyan Xu, Nobuhiro Nishio, Asahito Hama, Hiroshi Yagasaki, Yoshiyuki Takahashi, Koji Kato, Atsushi Manabe, Seiji Kojima, Jaroslaw P. MacIejewski

研究成果: ジャーナルへの寄稿学術誌査読

63 被引用数 (Scopus)

抄録

Summary Mutations in NF1, PTPN11, NRAS, KRAS and CBL have been reported to play a pathogenetic role in juvenile myelomonocytic leukaemia (JMML), a rare myelodyplastic/myeloproliferative neoplasm occurring in children. Recently, mutations in ASXL1 were identified in chronic myelomonocytic leukaemia and other myeloid malignancies. We sequenced exon 12 of ASLX1 in 49 JMML patients, and found 2 novel heterozygous (nonsense and frameshift) mutations, one occurring as a sole lesion, the other was in conjunction with a PTPN11 mutation. ASXL1 cooperates with KDM1A in transcriptional repression and thereby ASXL1 mutations may synergize with or mimic other JMML-related mutations.

本文言語英語
ページ(範囲)83-87
ページ数5
ジャーナルBritish Journal of Haematology
150
1
DOI
出版ステータス出版済み - 7月 2010
外部発表はい

!!!All Science Journal Classification (ASJC) codes

  • 血液学

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